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Modern molecular studies have furnished numerous insights into the processes that govern the event and progression of CLL, like numerous novel mutated genes clustered in various practical pathways. The CLL epigenome is reprogrammed from the modulation of regulatory regions that look de novo

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translocations or amplifications along with the genomic alterations now present in the initial CLL, but lack the prevalent mutations observed in Most important DLBCL indicating they might correspond to another biological classification.

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